• FHR Variability Categories

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    • Fetal heart rate is constantly varying from the baseline; this variability reflects a healthy fetal nervous system and cardiac responsiveness.
    • These fluctuations are characterized as absent if there is no variation in the amplitude range, minimal if fluctuation is less than 5 bpm, moderate if fluctuation is 6 to 25 bpm, and marked if fluctuation is greater than 25 bpm.
    • Absent variability indicates fetal academia but marked, moderate, and even minimal variation rules it out.
    • Conditions like fetal hypoxia, congenital heart anomalies, and fetal tachycardia can cause a decrease in variability.
  • Non-reassuring FHM

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    • One risk of fetal heart monitoring technology is false-positive results.
    • In fact, “nonreassuring” FHM strips, or those which show either late decelerations, or reduced beat-to-beat variability, or both, have a greater than 99% false-positive rate in predicting cerebral palsy.
  • ACOG Criteria

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    • In 2003, the American College of Obstetrics and Gynecology and the American Academy of Pediatrics recommended adoption of a definition of term intrapartum asphyxia developed by an international task force.
    • The definition requires that all four parameters must be met in order to diagnose intrapartum asphyxiation in a term fetus. These include metabolic acidosis, diagnosis of specific types of cerebral palsy correlated with the types of asphyxia damage seen in term fetal brains, early seizures and other neurological signs, and exclusion of all other causes.
    • There are other criteria that suggest an intrapartum timing of injury, nonspecific to asphyxia event. These include a hypoxic event immediately before or during birth, sudden, sustained bradycardia or loss of variability along with persistent, late, or variable decelerations, low APGAR scores (<3) beyond 5 minutes, multisystem organ involvement within 72 hours of birth, and early brain imaging showing acute, non-focal cerebral abnormalities.
  • Shoulder Dystocia

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    • In cases of shoulder dystocia, the upper fetal shoulder is impacted on the pubic symphysis, or more rarely, the lower shoulder becomes impacted on the sacral promontory or nonexible coccyx. In either case, this event prevents delivery of the baby. Shoulder dystocia can be a potentially catastrophic event since the fetal thorax is still within the pelvis and cannot properly expand for breathing.
    • While the rate of shoulder dystocia is higher with gestational diabetic women and macrosomic fetuses (>4500 grams at birth), most cases of shoulder dystocia occur with average-sized fetuses.
    • If a brachial plexus palsy (brachioplexopathy) occurs, it usually affects the portions of the brachial plexus that control the shoulder and elbow. Spontaneous recovery is the rule rather than the exception.
    • Most fetuses are successfully delivered with a combination of McRoberts maneuver and suprapubic pressure.
  • Shoulder Dystocia & Traction

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    • The uterus itself generates tremendous pressure during contractions. If the average systolic blood pressure (pressure generated by the left ventricle during a contraction) is 120 mmHg, the uterus alone generates more than 5 times that amount. When the accessory muscles (the diaphragm and abdominal muscles) are used to push in conjunction with contractions, this pressure increases to more than 10 times that amount.
    • The pull generated by a physician during downward traction for shoulder dystocia is slightly higher than the average systolic blood pressure and contributes only a very small amount to the total amount of generated pressures.
    • Uterine and abdominal pressures are good evidence that if shoulder dystocia is the cause of brachial plexus palsy, it is most likely from the intrinsic pressures of the uterus and body wall, not the caregiver.
  • Placental Abruption

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    • Abruption occurs when the placenta separates from the uterine wall. Most of the time, this is accompanied by vaginal bleeding as the blood travels between the membranes and the uterus. Some- times, however, there is no bleeding because edges of the placenta remain sealed.
    • Symptoms frequently include hypertonic uterus with severe abdominal pain and rapid contractions. If the abruption is large (more than about 50%), the fetus may not survive.
    • The specific cause of placental abruption is often unknown, but risk factors include abdominal trauma and maternal factors like smoking, drinking, and diabetes, among others.
    • Fetal distress occurs early in this condition in about half of all cases.
  • Carpal Tunnel Anatomy

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    • The carpal tunnel is a passageway on the palmar side of the wrist that houses the median nerve and finger flexor tendons. Anteriorly, the carpal tunnel is bordered by the transverse carpal ligament, a heavy band of fibers that forms the fibrous sheath containing the carpal tunnel; posteriorly, the carpal tunnel is bordered by carpal bones.
    • Guyon’s canal contains the ulnar nerve and artery; this anatomy does not pass through the tunnel, but lies superficial to it.
    • Carpal tunnel syndrome occurs when the median nerve, which controls sensations to the palm side of the thumb, first, second, and half of the third finger, becomes compressed at the wrist, within the carpal tunnel. This results in pain, weakness, or numbness in the hand and wrist, radiating up to the arm.
  • Carpal Tunnel Release

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    • If conservative treatment is not effective, surgical intervention may be chosen. Surgery can be done as an open procedure or endoscopically.
    • In endoscopic surgery, a small incision is made in the wrist and a dilator tube is inserted. An arthroscope with a camera is inserted in order to view the anatomy, then a cutting tool is used to sever the transverse carpal ligament in order to reduce pressure on the median nerve.
    • This type of surgery is less invasive and allows for faster recovery and less postoperative pain.

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